Table 1

Clinical features of MI caused by sVAD and non-sVAD

sVADN=47Non-sVADN=81P
Sex, N (%)
Female10 (21.28)12 (14.81)0.350
Male37 (78.72)69 (85.19)
Age, Med (IQR)44 (34,53)58 (52,66)0.000
Onset, N (%)
Sudden onset34 (72.34)73 (90.12)0.009
Non-sudden onset13 (27.66)8 (9.87)
Hypertension, N (%)21 (44.68)55 (67.90)0.010
Coronary heart disease, N (%)1 (2.13)5 (6.17)0.413
Diabetes, N (%)9 (19.15)37 (45.69)0.003
Dyslipidaemia, N (%)11 (23.40)26 (32.10)0.296
Atrial fibrillation, N (%)1 (2.13)2 (2.47)1.000
Past stroke history, N (%)1 (2.13)10 (12.35)0.054
Smoker, N (%)20 (42.55)27 (33.33)0.297
Alcohol, N (%)10 (21.28)10 (12.35)0.180
Family history of stroke, N (%)1 (2.13)5 (6.17)0.157
Minor neck injury, N (%)9 (19.15)1 (1.23)0.001
Headache, N (%)22 (46.81)6 (7.41)0.000
Neck pain, N (%)2 (4.26)4 (4.94)1.000
Side of infarction, N (%)
Left25 (53.19)37 (45.68)0.147
Right22 (46.81)38 (46.91)
Bilateral06 (7.41)
Medial MI3 (6.38)30 (37.04)0.000
Lateral MI43 (91.48)51 (62.96)0.000
Both medial and lateral MI1 (2.13)00.367
Vertebral perforative infarction, N (%)39 (82.98)62 (76.54)0.390
Multi-level medulla involvement, N (%)8 (17.02)19 (23.46)0.390
Cerebellum involvement, N (%)7 (14.89)13 (16.05)0.862
Involvement of other posterior vascular territories, N (%)2 (4.26)9 (11.11)0.326
  • MI, medullary infarction; sVAD, spontaneous vertebral artery dissection.